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MANEBMalawi National Examinations Board
MANEBInternational Academy of Ecology and Life Protection Sciences (Russian acronym)
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CUADRO 3 Plaguicidas detectados en sedimentos en microcuencas del Lago de Tota TABLE 3 Pesticides in sediments in microbasins of Lago de Tota Sistema Resultado Limite de Concentracion Lotico reporte (mg/kg) /metodo (mg/kg) Hato Ditiocarbamatos, 0.05/UV 0.12 Laguna Maneb, mancozeb, metiram, propineb, thiram y ziram.
No significant effect of [Na.sup.+], [K.sup.+], [Ca.sup.2+], [Mg.sup.2+], [Mn.sup.2+], [Fe.sup.3+], [Co.sup.2+], P[O.sub.4.sup.3-], S[O.sub.4.sup.2-], [Cl.sup.-], and N[O.sub.3.sup.-]-1000-fold, phenol and humic acid 10-fold, asulam, diazinon, malathion, maneb, nabam, phoxim, terbufos, thiabendazole, and thiram 50-fold was observed.
Andrade, "Chronic exposure to the fungicide maneb may produce symptoms and signs of CNS manganese intoxication," Neurology, vol.
(2012) also determined that mancozeb (it is a combination of Zinc and maneb) exposure can induce genotoxicity and apoptosis in cultured human lymphocytes.
Studies on the pesticides impact have shown that constant exposure to these substances could lead to disruptions of the thyroid [32] among which hypothyroidism is the most common abnormality after chronic exposure to benomyl, maneb and mancozeb [33].
Summary: Four novel maneb derivatives containing 1, 3, 4-thiadiazole were successfully synthesized and characterized by FT-IR, electrochemical analysis and 1H-NMR and 13C-NMR.
Neurotoxicant-induced animal models of Parkinson's disease: understanding the role of rotenone, maneb and PQ in neurodegeneration Cell Tissue Res 2004; 318(1):225-241.
(2010) had observed similar findings in rainbow trout exposed to sublethal concentrations of maneb and carbaryl.
Kosberg et al., "Combined exposure to Maneb and Paraquat alters transcriptional regulation of neurogenesis-related genes in mice models of Parkinsons disease," Molecular Neurodegeneration, vol.
Organic sulfur (dithiocarbamates) fungicides like zineb, maneb and heterocyclic nitrogen compounds gave adequate control against Colletotrichum gloeosporioides (Ruehl and Ledin, 1960).
Frank Soldner, research scientist in Jaenisch's lab and co-author of the study, said that exposing both normal and mutant neurons to pesticides, including paraquat, maneb and rotenone, created excessive free radicals in cells with the mutation, causing damage to dopamine-containing neurons that led to cell death.