MIP-2

AcronymDefinition
MIP-2Macrophage Inflammatory Protein-2
References in periodicals archive ?
Mice lacking IFN-[gamma] had elevated levels of IL-17 (p < 0.04), increased production of MIP-2 (p < 0.3), and accelerated corneal opacity [16].
While many of the above cytokines such as IL-17, IL-6, IL-1[alpha], and IFN-[gamma] and chemokines such as MIP-2, MCP-1, MIP-1a, and MIP-1[beta] have proinflammatory role in the destruction caused by HSV-1 including neutrophil infiltration and corneal inflammation, other chemokines and cytokines such as IL-10 and CCL3 can have a protective role.
Furthermore, IL-1 increases neutrophil infiltration into the cornea along with the concentration of MIP-2 and IL-6 [33].
Cytokines such as IL-17, IL-6, IL-1[alpha], and IFN-[gamma] and chemokines such as MIP-2, MCP-1, MIP-1[alpha], and MIP-1[beta] have proinflammatory role in the destruction caused by HSV-1 including neutrophil infiltration and corneal inflammation, and other chemokines and cytokines such as IL-10 and CCL3 can have a protective role.
b-LPS mediates the upregulation of chemoattractants and inflammatory cytokines, including MIP-2, MCP-1, TNF-[alpha], and IL-6, at the mRNA level in 3T3-L1 adipocytes.
b-LPS induce the upregulation of MIP-2, a strong chemoattractant of neutrophils, and MCP-1 in 3T3-L1 adipocytes (Figures 1 and 3).
In our study, the LPS derived from MDRA and MDRP increased the expression of MIP-2 in 3T3-L1 adipocytes.
coli-LPS (O111:B4), or saline and subjected to real-time PCR analyses to determine the mRNA expression levels of several adipokines, including MIP-2, TNF-[alpha], IL-6, MCP-1, TLR4, and eNOS.