The MMPH damage degrees were 17.8% for GI, 31.1% for GII, and 51.1% for GIII.
Furthermore, there was an early-stage increase in the BC volume in the MMPH and LMPH pathologies.
Relationships between the meniscus pathologies and the Baker's cyst volume Grade (G) % Mean [+ or -]SD P MMAH G1 48.8 12.9805 6.28513 G2 40 12.8139 7.98198 G3 11.1 18.7300 6.40197 p=0.229 MMPH G1 17.7 16.8313 8.51114 G2 31.1 14.2893 5.48554 G3 51.1 11.9639 7.31670 p=0.228 LMAH G1 60 11.4433 6.63048 G2 26.6 16.6250 7.48083 G3 13.3 16.9000 5.80331 p=0.048 LMPH G1 55.5 12.8712 7.37056 G2 35.5 14.9263 7.12328 G3 8.8 12.3175 6.18700 p=0.634 MMAH: Medial meniscus anterior horn; MMPH: Medial meniscus posterior horn; LMAH: Lateral meniscus anterior horn; LMPH: Lateral meniscus posterior horn Table 3.
The lungs, digestive system, retroperitoneum, and bone can be less frequently involved. The pulmonary manifestations of TSC include MMPH and lymphangioleiomyomatosis (LAM). MMPH in TSC was first described by Popper et al . in 1991.
TSC presented as MMPH was reported having better prognosis and specified treatment was usually unnecessary since the patients were usually asymptomatic and remained stable for a long time.
Multifocal micronodular pneumocyte hyperplasia, previously called atypical adenomatoid proliferations, used to be essentially diagnostic of tuberous sclerosis, but recent reports have shown MMPH can arise spontaneously.
The number of reported cases is small, and MMPH is seen most commonly in the setting of tuberous sclerosis.
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