5 In MSA-P
the degenerative changes predominantly affect the basal ganglia, particularly the putamen seen as hyperintense rim at the putaminal edge, atrophy and hypointensity of putaminal body on T2WI while in MSA-C changes predominantly affect infratentorial structures like pons and cerebellum, seen as atrophy and hyperintense signals in pons, cerebellum and middle cerebellar peduncles with pontine hyperintensity (hot cross bun sign) on axial image,4 which was seen in both of our patient's MRI.
As tonic chin EMG activity and AHI showed significant difference between MSA-P group and PD group, both of them had great associations with RBD, so we divided both groups into with (MSA + RBD; PD + RBD) or without RBD (MSA - RBD; PD - RBD) subgroups to make other comparisons.
Table 4] shows that the presence of MSA-P ([sz] = 0.
The main finding was that the MSA-P group had higher tonic tone than the PD group, irrespective of whether there was clinical RBD or not.
That was MSA-P group had higher tonic tone than the PD group, irrespective of whether there was clinical RBD or not.
The study showed higher tonic chin EMG density in the MSA-P group, which could be attributed to the more rapid and widespread neurodegenerative pathology in MSA-P as compared to that in PD.
We suspected that the SLD, PPN, and BG are affected earlier and more severely in MSA-P than PD, resulting in a higher REM sleep tonic chin EMG density in MSA-P.