MI-R

(redirected from Myocardial Ischemia and Reperfusion)
AcronymDefinition
MI-RMyocardial Ischemia and Reperfusion
References in periodicals archive ?
Caption: Figure 4: Myocardial ischemia and reperfusion injury.
Table 3: Hemodynamics during myocardial ischemia and reperfusion after 8 weeks of diet intervention.
Buja, "Myocardial ischemia and reperfusion injury," Cardiovascular Pathology, vol.
Neuhof, "Calpain system and its involvement in myocardial ischemia and reperfusion injury," World Journal of Cardiology, vol.
Di Meo, "Effects of myocardial ischemia and reperfusion on mitochondrial function and susceptibility to oxidative stress," Cellular and Molecular Life Sciences, vol.
The results from the present study identify a relation between HMGB1 and TLR2-signalling in myocardial ischemia and reperfusion.
In conclusion this study contributes to our understanding of the role of HMGB1 in myocardial ischemia and reperfusion. Besides the known involvement of its receptor RAGE, we here provide evidence for a role of TLR2 in mediating effects of endogenous HMGB1 released during acute myocardial ischemia and reperfusion.
Jiang, "ERS-PERK signaling pathway-mediated Nrf2/ARE-HO-1 axis: a novel therapeutic target for attenuating myocardial ischemia and reperfusion injury," International Journal of Cardiology, vol.
O'Connor et al., "Differential regulation of activator protein-1 and heat shock factor-1 in myocardial ischemia and reperfusion injury: role of poly(ADP-ribose) polymerase-1," American Journal of Physiology--Heart and Circulatory Physiology, vol.
Chen, "Biphasic modulation of the mitochondrial electron transport chain in myocardial ischemia and reperfusion," American Journal of Physiology--Heart and Circulatory Physiology, vol.
C I/R: control ischemia and reperfusion; DM I/R: diabetes rats subjected to myocardial ischemia and reperfusion; DM + EtOH I/R: diabetes + ethanol subjected to myocardial ischemia and reperfusion; DM + EtOH + CYA I/R: diabetes + ethanol + cyanamide subjected to myocardial ischemia and reperfusion; DM + EtOH + Atr I/R: diabetes + ethanol + atractyloside subjected to myocardial ischemia and reperfusion; DM + EtOH + Wor I/R: diabetes + ethanol + wortmannin subjected to myocardial ischemia and reperfusion.
It has been shown that oxidant-induced apoptosis after myocardial ischemia and reperfusion can be activated through translocation of NF-[kappa]B and stimulation of the release of TNF-[alpha] while application of postconditioning at the onset of reperfusion attenuated myocardial apoptosis through inhibition of NF-[kappa]B translocation [82].
Full browser ?