NALC

(redirected from N-acetyl-L-cysteine)
AcronymDefinition
NALCNational Association of Letter Carriers
NALCNorth American Lutheran Church (est. 2010)
NALCNational Law College (Nepal)
NALCNational Association of Local Councils
NALCNational Agricultural Law Center (University of Arkansas)
NALCN-acetyl-L-cysteine
NALCNippon Active Life Club (Japan)
NALCNational Advanced Leadership Camp
NALCNational Aging and Law Conference
NALCNational Association of Life Companies
NALCNaval Ammunition Logistics Center
NALCNaval Aviation Logistics Command
NALCNaval Aviation Logistics Center
NALCNew Applications License Charge (IBM)
NALCNavy Ammunitions Logistics Code
References in periodicals archive ?
in iron overload mice (Fe) significantly increased compared to the control group and were reversed by the administration of deferasirox (DFX) or N-acetyl-L-cysteine (NAC) in mean fluorescence intensity (MFI).
cells in iron overload (Fe) mice and after the administration of deferasirox (DFX) or N-acetyl-L-cysteine (NAC), in mean fluorescence intensity (MFI).
Results of colony-forming unit erythroid (CFU-E), granulocyte-macrophage (CFU-GM), burst-forming unit erythroid (BFU-E), and colony-forming unit mix (CFU-Mix) assays of bone marrow of iron overload (Fe) mice and after the administration of deferasirox (DFX) or N-acetyl-L-cysteine (NAC).
2011) Hydrogen and N-acetyl-L-cysteine rescue oxidative stress-induced angiogenesis in a mouse corneal alkali-burn model.
Oral N-acetyl-L-cysteine has been used to disrupt H.
Adjunctive, complementary interventions such as probiotics, antibiofilm enzymes, lactoferrin, N-acetyl-L-cysteine, and quercetin offer the opportunities to improve H.
N-acetyl-L-cysteine (NAC) is an antioxidant molecule endowed with immunomodulatory properties.
N-acetyl-L-cysteine inhibits primary human T cell responses at the dendritic cell level: association with NF-kappaB inhibition.
Does N-acetyl-L-cysteine influence cytokine response during early human septic shock?
Arsenic trioxide increased the cellular content of ROSs, especially hydrogen peroxide, and the antioxidant N-acetyl-L-cysteine.
N-Acetyl-L-cysteine prevents exercise-induced intestinal lymphocyte apoptosis by maintaining intracellular glutathione levels and reducing mitochondrial membrane depolarization.
N-acetyl-l-cysteine protects intestinal lymphocytes from apoptotic death after acute exercise in adrenalectomized mice.
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