OPIDPOrganophosphorus compounds Induce a Delayed Polyneuropathy
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OPIDP occurs in a small percentage of cases, roughly two weeks after exposure, where temporary paralysis occurs.
Titers of anti-NF, but not GFAP or MBP, NAb significantly correlated with changes in gait considered indicative of ataxia in OPIDP, decrease in stride length, and increase in width (Table 3).
OPIDP is a rare toxicity resulting from exposure to certain organophosphorus (OP) esters.
OPIDP is believed to be associated with exposure to combinations of a variety of OPs and other cholinesterase-inhibiting chemicals.
While the possibility remains that some acute OP poisoning may have occurred and resulted in OPIDP, a low level chronic poisoning would provide a better explanation of how so many service personnel came to be affected.
Secondly, does the spectrum of symptoms attributable to OPIDP correspond to the diversity of symptoms reported in veterans?
The balance of evidence suggests that the OPIDP model provides a general account of the variety of symptoms seen in GWS.