PAOPPulmonary Artery Occlusion Pressure
PAOPPeak Acid Output after Pentagastrin (gastroenterology)
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Right cardiogenic shock with low PAOP, typically from RV infarction, not a frequent cause, is dealt with apart in the FALLS-protocol (the usual practice of an ECG in a shock by the way simplifies the approach).
Baseline cardiorespiratory variables were measured 10 minutes before the propofol induction (T-10) and included: heart rate (HR; beats [minute.sup.-1]), systolic (SAP), diastolic (DAP) and mean arterial pressure (MAP; mmHg), CVP (cm[H.sub.2]O), CO (L [minute.sup.-1]), mPAP (mmHg) and PAOP (mmHg), respiratory rate ([f.sub.R]; breaths [minute.sup.-1]), body temperature (BT; [degrees]C) and arterial blood gas (partial pressure of oxygen--pa[O.sub.2] and carbon dioxide--paC[O.sub.2] (mmHg), pH, HC[O.sub.3.sup.-] (mEq [L.sup.-1]), base excess- BE (mEq [L.sup.-1]), and oxyhemoglobin saturation - Sa[O.sub.2] (%)).
Essentially, this was goal-directed therapy with supranormal haemodynamic targets that in order of priority, aimed for oxygen delivery of 550 to 600 ml/minute/[m.sup.2], a cardiac index of 3.5 to 4.5 l/minute/[m.sup.2], a mean arterial pressure greater than 70 mmHg, a PAOP greater of 18 mmHg, a heart rate of less than 120 bpm and a haematocrit greater than 27%.
The PAOP was obtained prior to the completion of an expiratory pause.
Patient data were recorded hourly and included heart rate, blood pressure, central venous pressure, PAOP, cardiac index (CI), D[O.sub.2] Sv[O.sub.2] and vasopressor or inotrope use.
And equally importantly if intensivists did reach a consensus of opinion, could the operators at the coalface consistently and accurately read the real PAOP in different patients' settings, e.g.
Mean arterial pressure (MAP, mmHg), central venous pressure (CVP, mmHg), mean pulmonary artery pressure (MPAP, mmHg) and PAOP (mmHg) were recorded with quartz pressure transducers.
As a result, pressure-based surrogates, such as central venous pressure (CVP) and pulmonary artery occlusion pressure (PAOP) have been measured clinically to assess volume status and response to fluid therapy (20).
In our report, during the first T-piece trial in all three patients, PAP and pulmonary artery occlusion pressure (PAOP) were both elevated (Table 1).
A pulmonary artery catheter was then introduced to monitor pulmonary pressures: pulmonary artery pressure (PAP) was 49/26 mmHg, mean 34 mmHg; mean occlusion pressure--(PAOP) was 13 mmHg and mixed venous saturation 73.7%.