PBECSPacific Basin Extended Climate Study
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found that both transcriptional and translational expressions of Wnt4 were upregulated in primary bronchial epithelial cells (PBECs) from COPD patients relative to control nonsmokers [55].
Such an upregulated Wnt5B and remodeling-related gene expression were also observed in the air-liquid interface- (ALI-) cultured PBEC cell model, particularly in the cells from COPD patients [50].
[16] showed that cigarette smoke extract (CSE) pretreatment of primary bronchial epithelial cells (PBECs) followed by P aeruginosa LPS stimulation reduced IL-8 release from COPD PBECs but increased it from cells of smokers without airflow obstruction and nonsmokers.