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References in periodicals archive ?
Caption: FIGURE 3: Colorectal adenocarcinoma (Caco-2) cells after 24-hour incubation with (a) cell culture medium and (b) cell culture medium spiked with 150 ng/ml phorbol 12-myristate 13-acetate (PMA).
AAPH: 2,27 -Azobis(2-methylpropionamidine) dihydrochloride C11-BODIPY: 4,4-Difluoro-5-(4-phenyl-1,3-butadienyl)-4bora -3a,4a-diaza-s-indacene-3-undecanoic acid DHR123: Dihydrorhodamine 123 DSI EC: Supplement Information Expert Committee EFSA: European Food Safety Administration FDA: Food and Drug Administration FRAP: Ferric reducing antioxidant potential GT: Green tea GTE: Green tea extract NEAC: Nonenzymatic antioxidant capacity PLIR: Peroxidation of leukocytes index ratio PMA: Phorbol 12-myristate 13-acetate Trolox: 6-Hydroxy-2,5,7,8-tetramethylchroman-2- carboxylic acid UA: Uric acid.
This NET release was similar to the one induced by cross-linking FcaRI [46] or by phorbol 12-myristate 13-acetate (PMA) stimulation [2] but different from the rapid, oxidant-independent NET release recently described [48].
[1] Nonstandard abbreviations: CRP, C-reactive protein; LOX-1, lectin-like oxidized LDL receptor; SR, scavenger receptor; DMEM, Dulbecco's modified Eagle's medium; FBS, fetal bovine serum; siRNA, small interfering RNA; PMA, phorbol 12-myristate 13-acetate; SRA-C6, anti-SR-A antibody; DAPI, 4',6-diamidino-2-phenylindole.
Phorbol 12-myristate 13-acetate (PMA) was also reported to stimulate the endogenous activator of protein kinase C (PKC), diacylglycerol (DAG) (Hong et al.
coli, phorbol 12-myristate 13-acetate (PMA), and formyl-methionyl-leucyl-phenylalanine (fMLP) in whole blood of healthy adults by the dihydrorhodamine flow cytometry assay.
In this study, we isolated phorbol 12-myristate 13-acetate (PMA) as an active component from Croton tiglium and investigated the action mechanisms in TLR signaling pathways.
Receptor-independent stimuli include phorbol 12-myristate 13-acetate (PMA) and certain long-chain unsaturated fatty acids (Leusen et al.
coli) as a particulate stimulus or the protein kinase C ligand phorbol 12-myristate 13-acetate (PMA) as a strong stimulus, granulocytes produced reactive oxygen metabolites.
Confluent NCI-H292 cells were pretreated with platycodin[D.sub.3] or deapi-platycodin for 30 min and then stimulated with PMA (phorbol 12-myristate 13-acetate) for 24 h.
Phorbol 12-myristate 13-acetate (PMA) was reported to stimulate the endogenous activator of protein kinase C (PKC), diacylglycerol (DAG) (Hong et al.
The same type of procedure was done in arteries maintained in [Ca.sup.2+]-free solution and contracted with phenylephrine (10 [micro]M) or phorbol 12-myristate 13-acetate (PMA; 10 [micro]M), allowing to observe the effect euxanthone in the contractions dependent on the release of the intracellular calcium stores or on the activation of PKC, respectively.