PDK

(redirected from Phosphoinositide-Dependent Kinase)
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AcronymDefinition
PDKPhi Delta Kappa (professional organization for teachers)
PDKPhosphoinositide-Dependent Kinase (biochemistry)
PDKPorsche Doppelkupplung (double clutch)
PDKPendidikan Dan Kebudayaan (Indonesian: Education and Culture)
PDKPartai Demokrasi Kebangsaan (Indonesian: National Democratic Party)
PDKProcess Design Kit
PDKPorsche Dual Klutch
PDKPartia Demokratike e Kosovës (Albanian: Democratic Party of Kosovo)
PDKPortal Development Kit (SAP Enterprise Portal)
PDKNational Democratic Party (former Yugolsav Republic of Macedonia)
PDKPeachtree-Dekalb Airport (Atlanta, GA, USA)
PDKPersonal Data Key (biometrics)
PDKPERL Development Kit
PDKPartia Demokristiane e Shqipërisë (Albanian Christian Democratic Party)
PDKPortable Deployment Kit
PDKPhosphatidylinositol-Dependent Kinase (immunology)
PDKPeripheral Developers Kit
PDKPortlet Developer Kit (Oracle)
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References in periodicals archive ?
In order to activate Akt, it requires that the phosphorylation of Thr308 in the activation loop by the phosphoinositide-dependent kinase 1 (PDK1) and Ser473 within the carboxyl-terminal (Figure 1).
Those involved in insulin signaling are the phosphoinositide-dependent kinase (PDK1), protein kinase B (PKB, a.k.a.
AS160: Akt substrate 160 kDa; GSK3: glycogen synthase kinase type 3; IRS: insulin receptor substrate; MAPK: mitogen-activated protein kinase; mTORC2: mechanistic target of rapamycin; PI-3K: phosphatidylinositol-3-kinase; PDK-1: phosphoinositide-dependent kinase; S6K: ribosomal protein S6 kinase.
It is also an initial attempt to understand the underlying mechanism of parasite clearance by macrophages following the expressions of some signaling molecules like phosphoinositide 3 kinase (PI3K), phosphoinositide-dependent kinase 1 (PDK1) and p38 mitogen-activated protein kinase (p38MAPK), respectively, in the groups of animals receiving therapy with attenuated homologous vaccine through I.M.
Molecularly, inflammation involves numerous intracellular signaling cascades, including the Src and Syk nonreceptor protein tyrosine kinases, phosphoinositide 3-kinase (PI3 K), phosphoinositide-dependent kinase 1 (PDK1), and Akt (protein kinase B) serine-threonine protein kinases, which contribute to the activation and upregulation of the transcription factors nuclear factor (NF)-[kappa]B and activator protein (AP)-1 [4, 5].
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