(redirected from Phosphoribosyltransferase)
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References in periodicals archive ?
Nicotinamide phosphoribosyltransferase (Nampt) may serve as the marker for osteoblast differentiation of bone marrow-derived mesenchymal stem cells.
Visfatin is also known as NAMPT (nicotinamide phosphoribosyltransferase), and NAMPT has the role as an NAD+-synthesizing enzyme within the cell (iNAMPT).
Imai, "The NAD biosynthesis pathway mediated by nicotinamide phosphoribosyltransferase regulates Sir2 activity in mammalian cells," Journal of Biological Chemistry, vol.
Chuang, "The relationship of visfatin/pre-B-cell colony-enhancing factor/ nicotinamide phosphoribosyltransferase in adipose tissue with inflammation, insulin resistance, and plasma lipids," Metabolism, vol.
Hypoxanthine guanine phosphoribosyltransferase (HPRT) served as a reference gene in this analysis (primer list).
Consistent with these findings, restoration of NAD+ levels by tanshinone II-induced nicotinamide phosphoribosyltransferase (Nampt) expression and increased AMPK activation resulted in reduced Tat-mediated HIV transactivation and reduced reactive oxygen species production [173].
Low levels of ATP from dietary restrictions or energy consumption induces nicotinamide phosphoribosyltransferase that generates [NAD.sup.+], which is a key factor for SIRT1 activation.
Visfatin is recognized as the formerly described Nicotinamide phosphoribosyltransferase (Nampt), and is a new adipokine which is a colony-stimulating factor activating the synthesis of pre-beta cells isolated from lymphocytes (7, 8).
albicans is primarily due to a decrease in the activity of the uracil phosphoribosyltransferase. [27-29] Our study showed 81% sensitivity to flucytosine which was similar to 63% by Bhatt et al.
The risk of developing PC is approximately 15% in patients with hyperparathyroidism-jaw tumor (HPT-JT) syndrome, an autosomal dominant disorder characterized by the germline mutation in the hypoxanthine phosphoribosyltransferase 2 (HRPT2), which encodes protein parafibromin.
Leucocytes are a major source of circulating nicotinamide phosphoribosyltransferase (NAMPT)/pre-B cell colony (PBEF)/visfatin linking obesity and inflammation in hu mans.
ACTB (b-actin) and HPRT (hypoxanthine phosphoribosyltransferase) primers, previously designed using predicted Vicugna pacos nucleotide sequences, were employed as internal control of qPCR (reference genes).