RIPKReceptor-Interacting Protein Kinase
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It has been reported that blockade of mTOR with its specific inhibitor CCI-779 stimulates autophagy and eliminates the activation of RIPKs in RCC4 cells [16].
In addition to the direct role, increased RIPKs expression may interact with other signals involved in cardiac hypertrophy.
These investigators demonstrated that palmitic acid-induced cardiomyocyte hypertrophy is associated with activation of necroptosis markers such as receptor interacting protein kinases (RIPK) 1 and 3.