RSV-A

AcronymDefinition
RSV-ARespiratory Syncytial Virus Subgroup A
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References in periodicals archive ?
The shifting in dominance from RSV-A to RSV-B every 2 years was repeatedly observed in our study and others (25).
Although RSV-A cases outnumbered RSV-B in our study, we did not determine the reason for this finding, whether RSV-A caused more symptomatic illness or transmitted more quickly than RSV-B among children.
We assigned 2013-14 as an RSV-B-dominant season for the purposes of modeling, although the numbers of RSV-A and RSV-B cases were almost equal.
For children with both RSV-A and RSV-B, the likelihood of having a length of stay of 3 or more days did not differ between those who had the single virus infection and those who were coinfected (48% vs.
Rhinovirus alone was associated with a lower chance of being hospitalized 3 or more days compared with RSV-A or RSV-B alone (OR 0.4).
Major Finding: Of 564 children infected with rhinovirus, 70% had RSV coinfection Rhinovirus alone was associated with a lower chance of being hospitalized 3 or more days compared with RSV-A or RSV-B alone (odds ratio 0.4).
A sublineage of RSV-A, noteworthy for an N276S substitution in the palivizumab binding site, emerged during 2008-2009 (8 [44%] of 18 RSV-A strains) and became the predominant RSV-A clade during 2009-2010 (25 [100%] of 25 strains) in palivizumab recipients and nonrecipients.
From 3 Canadian communities we detected a lineage harboring an N276S mutation in 44.4% of RSV-A sequences from 2008-2009 and 100% from 2009-2010, unrelated to palivizumab exposure.
Estimated mean nucleotide and amino acid identities of 170 unique respiratory syncytial virus fusion gene sequences * Mean nucleotide Sequence identity, % [+ or -] SD Overall, n = 170 89.59 [+ or -] 8.38 RSV-A (within group), n = 105 97.06 [+ or -] 1.33 RSV-B (within group), n = 65 98.65 [+ or -] 0.69 RSV-A vs.