rVSMC

(redirected from Rat Vascular Smooth Muscle Cell)
AcronymDefinition
rVSMCRat Vascular Smooth Muscle Cell
References in periodicals archive ?
Rat vascular smooth muscle cells were primary-cultured via explant method and grown in RPMI-1640 supplemented with 10% FBS, 100 [micro]g/mL penicillin, 100 [micro]g/mL streptomycin, and 2mM L-glutamine at 37[degrees] C in 5% C[O.sub.2] atmosphere.
LI, "Effect of calcitonin gene-related peptide on angiotensin II-induced proliferation of rat vascular smooth muscle cells," European Journal of Pharmacology, vol.
Liu, "Simvastatin inhibits homocysteine-induced CRP generation via interfering with the ROS-p38/ERK1/2 signal pathway in rat vascular smooth muscle cells," Vascular Pharmacology, vol.
We have investigated the effects and mechanisms and the receptor antagonist urantide of urotensin II (U-II) on the expression of U-II and its receptor GPR14 in rat vascular smooth muscle cells. U-II promoted the proliferation of vascular smooth muscle cells, influenced cell cycle, increased proliferation index and S-phase cell fraction, and dramatically promoted the expression of U-II and GPR14.
PKC may be involved in the post-translational modification of iNOS and in the regulation of the availability of iNOS substrates.14 Studies suggest that the MAP kinase pathway is partly involved in cytokine-induced iNOS expression in rat vascular smooth muscle cells (VSMCs).15
Differential signaling pathways in platelet-activating factor induced proliferation and interleukin-6 production by rat vascular smooth muscle cells. J Cardiovasc Pharmacol 1997; 30 : 169-75.
Nitric oxide generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells. J.
Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells. J Clin Investig 1989;83:1774-7.
We investigated the effects of Achillea millefolium extract in vitro on the growth of primary rat vascular smooth muscle cells (VSMCs) as well as the potential involvement of estrogen receptors (ERs) in this process.