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References in periodicals archive ?
The renal medulla will be the first affected part of the kidneys in the case of disrupted renal blood flow, and it is known that renal medullary oxygen tension is decisive for Pu[O.sub.2] (6).
Restoring renal blood flow to the affected kidney has been a theoretically attractive therapeutic option, considering that the central pathophysiology of ARAS involves decreased renal perfusion leading to activation of the renin-angiotensin-aldosterone system.
[1] It is more common among male infants with occlusion being unilateral more than bilateral with most common causes including prematurity, central venous lines, diabetic mother, asphyxia, infections, dehydration, polycythaemia, reduced renal blood flow due to shock and hyperosmolality, congenital heart disease and congenital deficiency of anticoagulants.
However, the effect of ET-1 antagonists in renal blood flow or renal fibrosis induced by the use of CsA in hypertensive animals was poorly investigated.
Therefore, close assessment of renal blood flow and perfusion represents an organ-directed target for hemodynamic therapy.
Turner, "The effect of arterial, venous, and arteriovenous occlusion on renal blood flow," Surgery, gynecology & obstetrics, vol.
Increased renal vascular contractility and resistance can lead to reduced renal blood flow and subsequent renal ischemia [3, 4].
For this, we monitored hepatic, small intestinal, and renal blood flow during elevated body temperature in sevoflurane anesthetized rats using a laser Doppler blood flowmeter.
First, consider the impact of metabolic acidosis on renal blood flow, glomerular filtration rate, and kidney size:
The kidney is a highly perfused organ, and the medulla receives only 5% of total renal blood flow. In addition to poor oxygen delivery, high oxygen demand by the medullary tubular cells assures that "normal" medullary P[O.sub.2] is always significantly lower than renal artery or vein oxygen levels.
Previous studies shows that patients undergone cardiac surgery usually have increased renal vascular resistance, and significant decrease (25% to 75%) in renal blood flow and glomerular filtration rate20,21.