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SPI1Salmonella Pathogenicity Island 1
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However, ample evidence indicated that the MeDEGs are involved in the characteristic pathology of SSc, including extracellular matrix remodeling (CSTA, CTSZ, CTSG, and ELANE), angiogenesis (CTSZ, CTSG, and CXCR6), coagulation/fibrolytic system (PLAUR and F2R), inflammatory response (C3AR1), and transcription factors associated with DEGs (NEF2, SPI1, and RUNX3) (Table 3).
Ito et al., "Association of a functional polymorphism in the 3;-untranslated region of SPI1 with systemic lupus erythematosus," Arthritis & Rheumatism, vol.
Los iniciadores utilizados para inducir la mutagenesis de los genes invG/invE (SPI-1) y ssaJ/ ssaK (SPI-2) y amplificar el gen de resistencia a la kanamicina del plasmido pKD4, se muestran en la tabla 2; estos se disenaron con secuencias homologas (36 nucleotidos) a la region del cromosoma adyacente a los genes objeto de delecion [H1 (invF) y H2 (invA) de SPI1 y H1 (ssal) y H2 (ssaL) de SPI2], de las SPI1 y SPI2 (numero de acceso GenBank para SPI1 U08280 y para SPI2 ssaL Y09357, ssaI AJ224892) mas la secuencia correspondiente a P1 y P2 del plasmido PKD4 (20 nucleotidos).
and Sa-Correia, I., 2003, "Adaptation of Saccharomyces cerevisiae to the herbicide 2,4-dichlorophenoxyacetic acid, mediated by Msn2p- and Msn4p-regulated genes: important role of SPI1", App.
Interfaces provided include a USB 2.0 full-speed connector, CAN routed to a three-pin terminal block, and [I.sup.2]C and SPI1 routed to UEXT.
PU.1, which is encoded by the gene SPI1 (spleen focus forming virus proviral integration oncogene 1), serves as a suppressor of acute myeloid leukemia, and its down-regulation results in an aggressive form of acute myeloid leukemia (15-17).
In contrast, the transcription factor SPI1 (spleen focus forming virus proviral integration oncogene), which is essential for the development of hematopoietic system (DeKoter and Singh 2000), is significantly upregulated in lymphocytes from study subjects with high levels of blood arsenic.
Salmonella entry into nonphagocytic epithelial cells requires several chromosomal genes (inv/spa) clustered in a pathogenicity island termed SPI1 (Salmonella pathogenicity island 1) (34).