SPSH

(redirected from Stroke-Prone Spontaneously Hypertensive)
AcronymDefinition
SPSHSouth Pacific Subtropical High (climate)
SPSHSupport Programme for Social Housing (South Africa)
SPSHSix-Phase Soil Heating
SPSHSchwippy Photo Scavenger Hunt
SPSHStroke-Prone Spontaneously Hypertensive
SPSHSaturday Photo Scavenger Hunt
References in periodicals archive ?
High-K diets reduce brain haemorrhage and infarcts, death rate and mesenteric arteriolar hypertrophy in stroke-prone spontaneously hypertensive rats," Journal of Hypertension, vol.
Reduction of cerebral infarction in stroke-prone spontaneously hypertensive rats by statins associated with amelioration of oxidative stress," Stroke, vol.
HMG-CoA reductase inhibitor has protective effects against stroke events in stroke-prone spontaneously hypertensive rats," Stroke, vol.
Isomura, "The stroke-prone spontaneously hypertensive rat: still a useful model for post-GWAS genetic studies?
Genetic mapping of a gene causing hypertension in the stroke-prone spontaneously hypertensive rat," Cell, vol.
Interaction between chromosome 2 and 3 regulates pulse pressure in the stroke-prone spontaneously hypertensive rat," Hypertension, vol.
Two genomic regions of chromosomes 1 and 18 explain most of the stroke susceptibility under salt loading in stroke-prone spontaneously hypertensive Rat/Izm," Hypertension, vol.
Contribution of genetic factors to renal lesions in the stroke-prone spontaneously hypertensive rat," Hypertension, vol.
Mitochondrial HMG-CoA synthase partially contributes to antioxidant protection in the kidney of stroke-prone spontaneously hypertensive rats," Nutrition, vol.
Identification of Stim1 as a candidate gene for exaggerated sympathetic response to stress in the stroke-prone spontaneously hypertensive rat," PLoS ONE, vol.
Yamori Y: The stroke-prone spontaneously hypertensive rat: Contribution to risk factor analysis and prevention of hypertensive diseases.
In this study, the effect of Choto-san was studied on NO function, hemorheological factors and endothelial function in stroke-prone spontaneously hypertensive rats (SHR-SP), which is a model of decreased cerebral circulation.