TAAD (%) = AA input - (AA output - EAAL) / AA input x 100
Emergent computed tomography (CT) showed a
TAAD with the primary entry tear at the inner curve of the ascending aorta and extending into the aortic arch.
Mutations in TGFBR2 have also been linked to familial thoracic aortic aneurysms and dissections (
TAAD), a syndrome associated with aneurysms of the ascending aorta and aortic dissections at relatively early stages of dilatation [54, 55].
As an example, Turner syndrome and
TAAD preferentially affect the thoracic aorta, while ADPKD may involve the thoracic, cervicocephalic, or intracranial vasculature.
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We describe a male patient with familial
TAAD, who does not have any features of the genetic syndromes associated with
TAAD.
In patients with
TAAD, we observed high levels of immune/inflammatory cells and a significant association of some inflammatory polymorphisms with the
TAAD susceptibility.
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PCT levels measured in patients undergoing Type A aortic dissection (
TAAD) were used to determine prognostic values for complications and surgical outcomes.
This study sought to investigate the association of circulating PTX3 levels with in-hospital mortality in patients with acute Type A aortic dissection (
TAAD).