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Average completion times showed the typical prolongation of TMT-B compared to TMT-A.
Figure 2 demonstrates a lesion subtraction analysis for TMT-B total errors.
A voxel-based lesion-behavior analysis revealed a significant association between voxel damage in the right hemispheric DLPFC and TMT-B total errors (cf.
Figure 3(b) depicts the voxels' location for which a voxel-based lesion-behavior analysis revealed a significant association between voxel damage and number of TMT-B shifting errors (cf.
Figure 3(c) depicts the voxels' location for which the voxel-based lesion-behavior analysis revealed a significant association between voxel damage and number of TMT-B sequencing errors (cf.
Voxel-based lesion-behavior analyses revealed no significant association between voxel damage and TMT-A or TMT-B completion time (cf.
Our results are congruent with Stuss et al.'s , according to which TMT-B errors, but not speed measures, are sensitive to DLPFC damage.
The TMT-B should be considered to be an endogenous task switching paradigm.
Our results show that TMT-B errors, but not completion times, are associated with DLPFC lesions.
We conclude that acute stroke lesions in the right hemispheric DLPFC (i.e., two regions within the right inferior frontal gyrus (BA9 and BA6)) are associated with enhanced TMT-B error proneness (i.e., increased TMT-B total and sequencing errors).
The empirical finding that associates right hemispheric DLPFC lesions with TMT-B errors needs explanation by a cognitive theory.
With regard to this issue it would be interesting to know to what extent the increased TMT-B error rate in our patients is explained partly by concurrent posterior cortical lesions causing a visuoperceptual deficit.
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