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References in periodicals archive ?
The level of TNF mRNA from the liver of D-GalN/LPS-treated group increased, and it reached at peak at 1 hr (p<0.01), and then persisted till 7 hrs (p<0.01) (Figure 3A), which was ~30-fold higher than the saline- or D-GaIN-treated group.
Tumor necrosis factor gene polymorphism results in high TNF level in sepsis and septic shock.
Long-term preincubation with low doses of TNF predominantly leads to the development of absolute tolerance [4].
The first decade of biologic TNF antagonists in clinical practice: lessons learned, unresolved issues and future directions.
Discovery of the causal role of the proinflammatory cytokine TNF in the pathogenesis of RA in the early 1990s (10), followed by clinical trials involving blocking antibodies to TNF, led to the development of the first biological therapies for RA (11, 12) and subsequently for CD (13).
Droge, "Divergent signalling via APO-1/Fas and the TNF receptor, two homologous molecules involved in physiological cell death," The EMBO Journal, vol.
The primary source of TNF alpha is macrophages and monocytes, other sources being T and B lymphocytes, natural killer (NK) cells, neutrophils, astrocytes, kupffer cells, fibroblasts, mast cells, smooth muscle cells, epidermal cells and endothelial cells.
Cutaneous adverse effects of TNF blockers i nclude infections, eczema and allergic skin reactions.
TNF-[alpha] also promotes apoptosis through binding to TNF receptor 1, resulting in many effects, ranging from inflammation to apoptosis [4].
The occurrence of tuberculosis (TB) in patients treated with immunosuppressive drugs has been recently highlighted by cases occurring in patients using anti-TNF alpha drugs, also called TNF inhibitors.