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Although it was demonstrated that in vitro T3 could enhance insulin-induced tyrosine phosphorylation of insulin receptor substrate by TRa1, TR expressions in the pancreas were not addressed in this study.
For the TRA1 region (1989, 1992, and 2004), TR82 region (1990, 1994, and 2000), TR33 region (1995, 2002, and 2008), and TR81 regions (1991, 1998, and 2009), it was noticed that the vulnerability indexes were equal to 1 in three occasions.
Saranteas et al., "Acute T3 treatment protects the heart against ischemia-reperfusion injury via TRa1 receptor," Molecular and Cellular Biochemistry, vol.
Diverse isoforms of TR genes (TRa1, TRa2, TRb1, TRb2, TRaa, TRab, TRba and TRbb) have been isolated from human (Weinberger et al., 1986), rat (Thompson et al., 1987), and fishes (Yamano et al., 1994; Liu et al., 2000; Marchand et al., 2001; Kawakami et al., 2003a, 2008; Nelson and Habibi, 2006, 2009; Masafumi et al., 2008; Galay-Burgos et al., 2008; Harada et al., 2008; Manchado et al., 2009; An et al., 2010).
All the translations were randomly assigned a sequential number, from TRA1 to TRA10, and all analyses were carried out using Wordsmith Tools.
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