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VCAM-1Vascular Cell Adhesion Molecule Number 1
References in periodicals archive ?
superoxide dismutase; GPx, glutathione peroxidase; SDS-PAGE, sodium dodecylsulfate polyacrylamide gel; SREBP, sterol regulatory element-binding protein; VCAM-1, vascular cell adhesion molecule 1; ICAM-1, intercellular adhesion molecule 1; NF-[kappa]B, nuclear factor-kappa B; PPAR[gamma], peroxisome proliferator-activated receptor gamma; MMP-9, matrix metalloproteinase 9; FRAP, ferric reducing/antioxidant power; AMPK, AMP-activated protein kinase.
Activin, a grape seed derived proanthocyanidin extract, reduces plasma levels of oxidative stress and adhesion molecules (ICAM-1, VCAM-1 and E-selectin) in systemic sclerosis.
Increased plasma soluble adhesion molecules; ICAM-1, VCAM-1, and E-selectin levels in patients with slow coronary sow.
2] Nonstandard abbreviations: CNS, central nervous system; VCAM-1, vascular cell adhesion molecule-1; EGF, epidermal growth factor; VEGF, vascular endothelial cell growth factor; VEGFR2, VEGF receptor 2; NSCLC, non--small cell lung cancer; bFGF, basic fibroblast growth factor; GFAP, glial fibrillary acidic protein.
Correlation (r) between soluble VCAM-1, e-Selectin and running speed (n=20).
In immunological and inflammatory reactions, ICAM-1 and VCAM-1 are surface glycoproteins that promote adhesion and subsequent recruitment of leukocytes [18].
The results showed that VCAM-1 levels were substantially elevated in the 60 individuals who underwent joint replacement, with the highest baseline levels seen in those who underwent bilateral joint replacement.
VCAM-1 is primarily induced by IL-4, and increased levels of both ICAM-1 and VCAM-1 have been found in tissue biopsy specimens of patients with allergic disorders.
The company reports the drug inhibits certain inflammatory proteins, including VCAM-1, endothelial cells lining the inside surfaces of blood vessel walls.
Polymerization of hemoglobin S, red cell sickling, and increased expression of VCAM-1 lead to vaso-occlusive phenomena in various organs, particularly the bone marrow and lungs.
The research specifically demonstrates the central role of VCAM-1 and VLA-4 in the survival and growth of metastatic breast cancer cells.
The levels of different cell adhesion molecules ICAM-1, VCAM-1, and E-selectin were analyzed for their association with the TEQ values using two different statistical models in linear regression.