VDCC

AcronymDefinition
VDCCVoltage-Gated Calcium Channel
VDCCVirus-Dependent Cellular Cytotoxicity
VDCCVoltage-Dependent Calcium Channels
References in periodicals archive ?
The time of application of five minutes after the SCI was chosen in order to early block the N-type VDCC and decrease injuries resulting from exaggerated influx of calcium and its deleterious effects (DASURI et al.
Board 18 AN EXAMINATION OF THE ROLE OF NMDA RECEPTORS AND VDCCS IN ACQUISITION AND RETENTION OF A TIMING TASK.
VDCC active element was implemented by another UCC-N1B circuit consisting of one CCII+/-(forming OTA part from Fig.
Yesil, "Positive/negative lossy/lossless grounded inductance simulators employing single VDCC and only two passive elements", AEU--Int.
Reference Active Mode of Available elements operation transfer (number of functions devices) [30] ZC-CG- VM HP iAP VDCC (1) DT [31] OTA (2), VM AP, LPZ, ECCII (1) iDT, AZ [32] OTA (2) VM AP, HP, LP [33] MCDU (1) CM AP, iHP, LP [34] MCDU (1) CM HP, LP, AP Reference Simulated (sim) Passive Tunable pass- and/or measured components band/stop-band (exp) (number) gain analysed [30] sim & C (1) exp no/no [31] sim C (1) no/no [32] sim C (1), R no/no (1) [33] sim & C (1) no/no exp [34] sim C (1) no/no TABLE II.
3]R, inositol 1,4,5-trisphosphate receptor; RyR, the ryanodine receptor; VDCC, voltage-dependent [Ca.
2000) have shown that animals in which VDCCs have been antagonized increase the number of reference memory errors (RMEs) they make after a 7-10 day period in which they have not been in the maze - animals treated with saline or the NMDAr antagonist, MK801, do not increase their errors.
Here we hypothesized that blocking the NMDARs would impair short-term retention (24 hours), and blocking the VDCCs would impair long-term retention (10 days) in a fear conditioning paradigm where context and tone are paired with mild footshock.
Ca] channels appear not only to integrate the output from several signaling cascades but also arrest VDCC activation to relax SMCs.
In this study, we provided the first evidence that the vasorelaxant effects of baicalin could be mainly attributed to VDCC inhibition and [BK.
Ca]-channel inhibitor IbTX, VDCC activator Bay K8644, and PKC activator PMA-induced contractions at concentrations [greater than or equal to] 30 [micro]M.