VLA4Very Late Antigen 4
VLA4Anti-Very Late Antigen 4
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References in periodicals archive ?
Javor et al., "VLA4 gene polymorphism and susceptibility to multiple sclerosis in Slovaks," Folia Biologica, vol.
Despite the identification of the major adhesion molecule interactions responsible for NK cell binding to pECs and the availability of commercialized monoclonal Abs blocking, for example, VLA4 (natalizumab), there are essentially no studies trying to test adhesion blocking approaches invivo or ex vivo [14,115].
Macrophages promote atherosclerosis via production of various key biomediators (3), including cytokines such as interleukin (IL)-[beta], [1] tumor necrosis factor-[alpha] (TNF-[alpha]), and IL-6; chemokines such as IL-8 and monocyte chemoattractant protein-1 (MCP-1); matrix metalloproteinases; and integrins (CD11b and VLA4).