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References in periodicals archive ?
For bulimia nervosa, consider a combination of SSRI and psychotherapy.
For bulimia nervosa, lifetime prevalence is 1 to 4.
This is the first study that suggests that brain dopamine related reward circuitry, pathways that modulate our drive to eat, may have a role in bulimia nervosa.
To compound the problems of high comorbidity and role impairment, fewer than half of individuals with bulimia nervosa seek treatment.
2 percent of females experience bulimia nervosa at some point in their lives.
Bilateral parotid enlargement as a consequence of bulimia nervosa has been well documented in the literature (1-4) Although the exact mechanism is not well understood, several hypotheses exist.
2008) constataram que as mulheres com bulimia nervosa antes e durante a gravidez exibiam padroes dieteticos diferentes das gravidas sem perturbacoes do comportamento alimentar.
Persistent alterations in behavior and serotonin activity after recovery from anorexia and bulimia nervosa.
The National Comorbidity Survey Replication Adolescent Supplement (NCS-A) showed suicidality most prevalent in adolescents with bulimia nervosa (53% had suicide ideation, 25.
A Study About Anorexia and Bulimia Nervosa in University
The DSM IV diagnostic criteria for bulimia nervosa is as follows (8):
Bulimia nervosa is characterized by binge eating and inappropriate compensatory behaviour such as fasting, vomiting, using laxatives, or exercising to prevent weight gain [5,6].