There are means to confirm the results obtained with the auditory training, as the auditory middle latency responses (AMLR), which is an objective test, being the most indicated in the identification of
Central Auditory Nervous System (CANS) alteration, which generates a series of positive and negative waves between 10 and 80 ms after the beginning of the acoustic stimulation, presenting a negative wave in approximately 20 ms and a positive peak around 30 ms [16, 17].
However, early exposure to sound environment induced effects on the cochlea that were different from the plasticity in the
central auditory nervous system during development.
According to Balbani and Montovani (2003), the fluctuating nature of hearing loss in otitis media (which can alternate with periods of normal hearing) leads to an inconsistent sound stimulation of the
central auditory nervous system, hindering the children from perceiving the speech sounds.
(10) The ability to locate the spatial origin of a sound source requires the capacity of the
central auditory nervous system to detect and compute a number of acoustic cues such as small differences in the arrival time and intensity of signals reaching the 2 ears.
Occupational exposure to toxic agents may induce adverse effects on human hearing, that are associated with cochlear pathology and dysfunction of the
Central Auditory Nervous System (CANS).
The
central auditory nervous system (CANS) is a maturing system and typically reaches adult-level function around 10 to 11 years of age.
While peripheral auditory dysfunction is commonly associated with traumatic injury due to blast exposure [1-8], there also is an increasing awareness of the vulnerability of the
central auditory nervous system (CANS) to blast-induced injury [9-11].
