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References in periodicals archive ?
The research study provides estimates for Vitamin B12 (Cobalamin, Cyanocobalamin) Forecast till 2025*.
Clinical and molecular heterogeneity in patients with the cblD inborn error of cobalamin metabolism.
The researchers' hope was that by giving injections of cobalamin, the cats' levels of cobalamin (and secondarily, levels of MMA) would normalize and the signs of gastrointestinal illness would improve.
It has been suggested that vitamin B12 deficiency may be treated by parental or high dose oral cobalamin therapy (Stabler, 2013).
Nitrous oxide "whippit" abuse presenting with cobalamin responsive psychosis.
Concentrations of MMA and tHcy were similar to those observed for several patients with inherited cobalamin C defect followed in our laboratory; therefore, the possibility of an intracellular cobalamin defect was initially raised and communicated to the attending pediatric neurologist.
Vitamin B12 status was assessed in infancy with the use of plasma cobalamin, total homocysteine (tHcy), and methylmalonic acid (MMA).
Since p.Tyr130 maps in the conserved cobalamin binding motif (122-HXXGX126-154GG156), the p.Tyr130His mutation may affect clb binding or protein structural integrity and it could be associated with a late onset phenotype.
Cobalamin is an essential water soluble vitamin [8] required as a coenzyme for methionine synthase and methylmalonyl-CoA mutase [4], in the methylation of DNA and the Krebs cycle, respectively.
Chief among the aforementioned influences was a deficiency of essential microelements and vitamins in the preconception period, such as the B-vitamins (i.e., folic acid and cobalamin).
Cobalamin (vitamin B12) deficiency induced TMA is a rare condition which closely resembles the clinical features of TTP such as thrombocytopenia, hemolytic anemia, and schistocytosis.