e-NOSEndothelial Isoform of Nitric Oxide Synthase
References in periodicals archive ?
Whether EOAZ is directly interacting with endothelial NOS (e-NOS) or with other factors, which may increase the e-NOS activity remains to be further investigated.
Testosterone levels and e-NOS were monitored along with routine clinical chemistry.
Endothelial injury and stress, decreased bioavailability of nitric oxide, oxidative stress leading to the formation of toxic peroxy nitrite, association of hyperhomocystinemia with higher circulating levels of asymmetric dimethyl arginine (ADMA) which inhibit the production of nitric oxide and promote uncoupling of e-NOS, protein modification by S-thiolation, and decreased expression of thrombomodulin in aortic endothelium leading to functional protein C deficiency have all been implicated to a varying degree.