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Related to glomerulosclerosis: focal segmental glomerulosclerosis
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References in periodicals archive ?
Celiac disease and focal segmental glomerulosclerosis: Is it a fortuity finding?
Glomeruli are functional filtration units comprising a capillary network of endothelial cells and mesangial cells, which are separated from podocytes by a basement membrane.[17],[18] Podocyte injury and loss contribute to proteinuria and glomerulosclerosis.[7],[19],[20],[21],[22],[23] Numerous podocyte gene products, such as nephrin (NPHS1),[24] podocin (NPHS2),[25] laminin beta-2 (LAMB2),[26],[27] a-actinin-4 (ACTN4),[28] Wilms' tumor suppressor gene 1 (WT1),[29] and inverted forming 2 (INF2),[30] are required to construct the podocyte body and foot processes.
Angiotensin antagonists and steroids in the treatment of focal segmental glomerulosclerosis. Semin Nephrol 2003; 23(2): 219.
The biopsy results showed tubulointerstitial disease in 3, collapsing glomerulopathy in 2 and interstitial nephritis with glomerulosclerosis in 1 patient.
FSGS: Focal segmental glomerulosclerosis GalT-KO: Alpha-1,3-galactosyltransferase knockout HAR: Hyperacute rejection h: Human hDAF: Human decay-accelerating factor MCD: Minimal change disease MHC: Major histocompatibility complex NHP: Nonhuman primates PBL: Peripheral blood lymphocytes POD: Postoperative day SMPDL-3b: Sphingomyelin phosphodiesterase acid-like 3b Tg: Transgenic VTL: Vascularized thymic lobe XKTx: Xenogeneic kidney transplantation.
We have shown that NTN mice develop a chronic stage of kidney disease within 21 days as seen by glomerulosclerosis, fibrosis, inflammation, tubular damage, elevated systemic markers of kidney damage, and albuminuria, whereas, for example, classical diabetic nephropathy models develop mild signs of CKD within 15-18 weeks [7].
Ghiggeri, "New insights into the pathogenesis and the therapy of recurrent focal glomerulosclerosis," American Journal of Transplantation, vol.
We report an atypical case of hepatic glomerulosclerosis showing progressive loss of renal function together with nephrotic syndrome and active glomerular lesions.
Wong, "Novel Homoplasmic Mutation in the Mitochondrial tRNATyr Gene Associated with Atypical Mitochondrial Cytopathy Presenting with Focal Segmental Glomerulosclerosis," American Journal of Medical Genetics, vol.
A subsequent renal biopsy showed focal segmental glomerulosclerosis (FSGS) in two of twelve viable glomeruli, with tuft-capsule adhesion, hyalinosis, segmental intracapillary hypercellularity, and segmental splitting of capillary walls, predominantly in regions of segmental sclerosis.
We report the successful surgical management of a 28-year-old male with EPS s/p kidney transplant in 2015 for end-stage renal disease (ESRD) secondary to focal segmental glomerulosclerosis (FSGS).
Only 40 patients (48%) had abnormal histological changes, which included glomerulosclerosis (GS, 25%),interstitial fibrosis (IF, 13%), acute tubular necrosis (ATN, 5%), and focal tubal atrophy (FTA, 5%) (Table 1).