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hK1Human Kallikrein 1
hK1Human Keratin 1
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References in periodicals archive ?
According to the [Chi.sup.2] statistic, the symmetry hypothesis is not rejected for the variables: fixed capital formation (Kpc), human capital (HK1) and CO2 emissions in the first equation, and for the variable HDI in the second equation, implying that these variables should be expressed in first differences in the estimated models.
h) Somatic overexpression of Hexokinase 1 (HK1) and CHI
Caption: FIGURE 1: Cell viability of NPC cells, HK1 treated with 1a-d and 2a-d, aspirin, and standards (cisplatin and 5-fluorouracil).
schlegelii SZG9 SZG2 J052 J053 J049 J054 J048 HK2 HK1 SZG6 SZG3 SAR003 SAR002 SAR022 PM016 PM019 PM020 SUM037 SUM038 SUM036 PM010 SZG4 SAR023 PM015 PM009 SAR021 SAR005 SAR004 SZG1 HK3 SZG10 HK4 SZG5 J045 SZG7 PM008 SZG8 PM007 J047 PM012 PM014 PM017 PM018 T.
First, it performs inhibition of hexokinases HK1 and HK2 and phosphoglycerate mutase (PGAM), which catalyze the first and eighth glycolysis steps [104, 105].
Six NPC-derived cell lines (HONE-1 [28], SUNE-1 [29], HK1 [30], TW01 [31], TW04 [31], and C666-1 [32]) and an immortalized nonmalignant nasopharyngeal epithelial cell line (NP69 [33]) were used in this study.
Each image is decomposed into two low frequency coefficients (La1, La2) to (Lk1, Lk2) and some high frequency coefficients (Ha1, Ha2, ..., Han) to (Hk1, Hk2, ..., Hkn).
Nasopharyngeal carcinoma cell lines used were SUNE1, HONE1, HK1, and TW01, and nonmalignant nasopharyngeal epithelial cell line was NP69 [9].
Innovia Films will be exhibiting its new com post able Na ture Flex[TM] 55 HK1, a hermetically sealing film providing an effective barrier against mineral oil residues.
At about the time of the Phalcon-deal cancellation and the Einstein impasse, China signed an agreement of almost equal value to the Phalcon contract for Israeli-made HK1 and 2 satellites to broadcast the 2008 Olympic Games in Beijing.
For instance, the up-expressed miR-143 and miR-138 can, respectively, target the genes, HK2 and HK1, which are the crucial enzymes in glycolysis and so that lead to potential glycemia [28, 29]; miR-9 and miR-204 were reported that they can regulate the insulin secretion by targeting the gene, SIRT1 [30-32], while miR-96 can decrease the expression of NOC2 which is involved in the insulin secretion [33].
In normal tissues, this crucial step is catalyzed by four different HK isoforms (HK1, HK2, HK3, and glucokinase) indicating that regulation of glucose phosphorylation can vary in different tissues under different condition [140].