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Related to luteinizing hormone-releasing hormone: luteinizing hormone-releasing hormone agonist
LH-RHLuteinizing Hormone-Releasing Hormone
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Presence of luteinizing hormone-releasing hormone (LHRH) mRNA in rat spleen lymphocytes.
When luteinizing hormone-releasing hormone agonists are first prescribed, there is a temporary flare of testosterone production that may last for 30 days before testicular testosterone production shuts down.
Influence of food restriction on neuropeptide-Y, proopiomelanocortin, and luteinizing hormone-releasing hormone gene expression in sheep hypothalami.
Modern drug therapy is as effective as surgery, however, and is more acceptable to most patients.[1] Estrogens such as diethylstilbestrol (DES) are effective, but are associated with an increased risk of thromboembolism and cardiovascular complications.[2,3] Luteinizing hormone-releasing hormone (LHRH) analogs produce response rates that are comparable to those seen with DES and orchiectomy but without the cardiovascular complications or potential psychological distress.
The brain starts to secrete a hormone called luteinizing hormone-releasing hormone (LHRH) in periodic bursts, which causes the pituitary gland to release hormones called gonadotropins.
Failure to maintain a suppressed level of serum testosterone during long-acting depot luteinizing hormone-releasing hormone agonist therapy in patients with advanced prostate cancer.
Initiation and progression of puberty are controlled by signals from the central nervous system that stimulate the pulsatile diurnal secretion of luteinizing hormone-releasing hormone (LHRH) from the hypothalamus into the hypothalamic--pituitary portal system (Sarkar and Fink 1979; Sarkar et al.
Active immunization of heifers against luteinizing hormone-releasing hormone, human chorionic gonadotropin and bovine luteinizing hormone.
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