NADH

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Related to nicotinamide adenine dinucleotide: nicotinamide adenine dinucleotide phosphate, adenosine triphosphate
AcronymDefinition
NADHNicotinamide Adenine Dinucleotide
NADHNorth American Digital Hierarchy
NADHNicotinamide Adenine Dinucleotide plus Hydrogen
References in periodicals archive ?
Fluorescence Signal in the Experiment Channel Wavelength Color Name Range, nm Code Dominant Sources SHG 360-400 Red Collagen SWAF 420-490 Green NADH, lipofuscin LWAF 550-650 Blue FAD, NADH, lipofuscin, iron, etc Abbreviations: FAD, flavin adenine dinucleotide; LWAF, long-wave-length autofluorescence; NADH, reduced nicotinamide adenine dinucleotide; SHG, second harmonic generation; SWAF, short-wave-length autofluorescence.
Koshland Jr, "Structure of isocitrate dehydrogenase with isocitrate, nicotinamide adenine dinucleotide phosphate, and calcium at 2.5-A resolution: a pseudo-Michaelis ternary complex," Biochemistry, vol.
In this enzymatic method, hexokinase converts glucose to glucose-6-phosphate, which is then oxidized with concurrent reduction of nicotinamide adenine dinucleotide phosphate.
GSH: glutathione; GSSG: glutathione disulfide; NAD: nicotinamide adenine dinucleotide; NADH: oxidoreductase-induced nicotinamide adenine dinucleotide; NADP: nicotinamide adenine dinucleotide phosphate; NADPH: oxidoreductase-induced nicotinamide adenine dinucleotide phosphate.
Hyperglycemia can lead to the production of ROS such as superoxide, hydrogen peroxide, and hydroxyl radicals during a number of enzymatic and nonenzymatic pathways such as glucose oxidative phosphorylation, the polyol pathway, advanced-glycation end-products, outflow during mitochondrial respiratory chain, and nicotinamide adenine dinucleotide phosphate oxidase activation.
Methemoglobinemia is an uncommon condition caused by induction of several toxic compounds including chlorates, inorganic, and organic nitrates or with drugs such as local anesthetics and sulfa drugs including dapsone or it can be congenital due to of red cell nicotinamide adenine dinucleotide reductase deficiency.
Our landmark research found that a deficiency in a vital molecule known as NAD (nicotinamide adenine dinucleotide) can prevent a baby's organs from developing correctly in the womb in some cases.
Endari reduces oxidant damage to red blood cells by improving the redox potential of nicotinamide adenine dinucleotide (NAD), a coenzyme that has been identified as the primary regulator of oxidation.
By reducing nicotinamide adenine dinucleotide phosphate (NADP) to nicotinamide adenine dinucleotide phosphate hydrogen (NADPH), G6PD allows the cells to maintain glutathione in its reduced form.
Nicotinamide adenine dinucleotide ([NAD.sup.+]) is a vital cofactor for metabolizing energy and a substrate for various enzymes [13].
Nicotinamide phosphoribosyltransferase (NAMPT) is the rate-limiting enzyme for biosynthesis of nicotinamide adenine dinucleotide (NAD+) [1].
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