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In our research, we found the unidirectional changes of serum concentration of sCD25, sCD38, and sCD8 dependent on disease severity in patients with COPD.
In GOLD II patients, the serum levels of sCD25, sCD38, and sCD8 molecules in the exacerbation and in the stable phase exceeded the biomarker values of healthy nonvolunteers.
Since sCD25, sCD38, and sCD8 are produced by activated mononuclear cells, their content in the biological fluids is considered as an indirect marker of immune system cell activation [15, 16].
The function of sCD38 molecules is also targeted to block the immune-regulatory activity of membrane protein form [32-34].
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