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SAT area and circulating concentrations of hsCRP, sTNFRI, sTNFRII, sCD163, sCD14, and leptin did not differ significantly by metabolic health status among HIV-uninfected obese men.
SAT area and circulating IL-6, hs-CRP, sTNFRI, sTNFRII, CD163, sCD14, and leptin concentrations did not differ by metabolic health status among HIV-infected obese men.
Among normal weight metabolically healthy men, the HIV-infected men had higher concentrations of IL-6 (p = 0.03), hs-CRP (p = 0.001), sTNFRII (p = 0.001), CD163 (p = 0.002), CD14 (p <0.001), and HOMA-IR (p <0.001) and lower concentrations of adiponectin (p = 0.02) and greater VAT area (p = 0.01) and less SAT area (p <0.001) than their HIV-uninfected counterparts (Table 4).
Among normal weight metabolically unhealthy men, the HIV-infected men had higher concentrations of sTNFRI (p = 0.003), sTNFRII (p = 0.001), CD163 (p = 0.013), CD14 (p <0.001), greater VAT area (p = 0.024), and less SAT area (p <0.001) than their HIV-uninfected counterparts.
Among HIV-infected men with normal weight, lower log hs-CRP, sTNFRI, sTNFRII, and HOMA-IR values and higher adiponectin concentrations were associated with metabolic health (Table 5).
After a Bonferroni correction for multiple testing, significant correlations were observed between GDF15 levels and age, as well as circulating levels of LCN2 and various inflammatory and oxidative stress markers, including sE-selectin, sVCAM1, sICAM1, sTNFRII, and homocysteine levels (Table 4).
We confirmed that genetic polymorphisms around the GDF15 gene were associated with GDF15 levels and also that GDF15 levels were associated with age, smoking, hypertension, and DM as well as circulating levels of LCN2 and many inflammatory and oxidative stress biomarkers including sTNFRII and homocysteine that have not been previously reported.
In a relatively young population, we further found a significant association between GDF15 and other inflammatory and oxidative stress markers including sTNFRII and homocysteine in our Taiwanese cohort.
 established a correlation between TNF[alpha] and sTNFRI levels but not with sTNFRII and Koga et al.
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