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References in periodicals archive ?
Inhibiting Tyrosine Phosphorylation of NR2B Ameliorated Hyperalgesia on the Mechanical Allodynia Induced by Chronic Migraine.
The concomitant tyrosine consumption was 1.84 mg/ml, a ~40% increase in comparison to control which was highly significant (pless than 0.05).
In case of pleural effusion caused by tyrosine kinase inhibitors, treatment should be performed according to degree of effusion.
Furthermore, there are three small-molecule inhibitors of the tyrosine kinase domain (TKIs) that interfere with the binding of ATP, also approved to treat non-small cell lung cancer (i.e., gefitinib, erlotinib, and lapatinib) [4].
The significance of this process in cancer cells became evident when they identified a tyrosine kinase, WEE1, as being a critical regulator of the process.
Of special interest to the researchers were: 1) the mice's locomotor activity in response to cocaine; 2) the degree of conditioning for place preference; 3) electrical activity of the ventral tegmental area (part of the brain's reward system; it plays a role in sense of euphoria and well-being); 4) the presence of tyrosine hydroxylase in the VTA; and 5) the presence of deoxyribonucleic acid (DNA) sequences for the Clock gene in the brain.
KEY WORDS: American lobster, Homarus americanus, alkylphenols, hisphenol A, shell hardening, sclerotization, tyrosine, molting, lobster, cuticle
His urine contained a large amount of 4-hydroxyphenyllactate (4-HPL) and a small amount of 4-HPP, 4-hydroxyphenylacetate (4-HPA) and tyrosine. The authors proposed, on the basis of the physical findings and biochemical observations, that the patient might have another type of inborn error of tyrosine metabolism, different from that in Medes's case.
Ror1 is a receptor tyrosine kinase with an intracellular catalytic domain.
A team of researchers led by Jeffrey Engelman, at Massachusetts General Hospital Cancer Center, Boston, has now identified a potential way to effectively use receptor tyrosine kinases inhibitors to treat individuals with KRAS mutant colorectal cancers -- combine them with inhibitors of the MEK/ERK signaling pathway.
Studies in our lab have demonstrated that oligomeric A[beta] stimulates microglia cultures through activation of tyrosine kinases such as Lyn.
Sperm capacitation in mammals has been showed to correlate with a series of changes including alterations in membrane fluidity (Harrison and Miller, 2000), cAMP concentration (Visconti et al., 1997, 1998), activation of PKA (Lefiever et al., 2002), acquisition of a special motility known as hyperactivation (Si and Okuno, 1999) and increase in protein tyrosine phosphorylation (Visconti et al., 1995b).