Studahl, "Varicella-zoster virus
infections of the central nervous system--prognosis, diagnostics and treatment," Journal of Infection, vol.
Fukuda et al., "Detection of varicella-zoster virus
DNA in human geniculate ganglia by polymerase chain reaction," Journal of Infectious Diseases, vol.
A similar case report by Pui JC (8) detected the varicella-zoster virus
in the muscularis propria layer of the terminal ileum in an HIV-positive patient who presented with the signs and symptoms of intestinal pseudo-obstruction.
Multiplex polymerase chain reaction for the detection of herpes simplex virus, varicella-zoster virus
and cytomegalovirus in ocular specimens.
Neurologic complications of the reactivation of varicella-zoster virus
. N Engl J Med 2000;342:635-45.
He examined data from 25 earlier studies on varicella-zoster virus
in a variety of countries around the world, and plotted these data against a range of climatic factors, the BBC reported.
After a bout of chickenpox, the varicella-zoster virus
lies dormant in the body, but as immunity wanes with age, the virus can reactivate in the spinal nerves leading to the skin.
Chickenpox is caused by primary infection with the varicella-zoster virus
(VZV), which can remain dormant in nerve tissue.
To the Editor: Varicella-zoster virus
(VZV), a human herpesvirus, is the causative agent of varicella (chickenpox) and herpes zoster (shingles).
Nearly equal proportions of cases and controls (76%) recalled having had primary infection with varicella-zoster virus
(VZV) is the etiological agent of primary varicella (chicken pox) in childhood; it establishes a latent infection that may be reactivated to cause herpes zoster (shingles).